Aspirin lowers thromboxane production by inhibiting cyclooxygenase, which reduces platelet aggregation and the risk of arterial thrombosis.
阿司匹林通过抑制环氧化酶降低血栓烷的生成,从而减少血小板聚集并降低动脉血栓风险。
Robbins and Cotran Pathologic Basis of Disease(《罗宾斯与科特兰病理学基础》):在血栓形成、血小板活化与血管反应章节中常讨论 TXA₂。
Goodman & Gilman’s The Pharmacological Basis of Therapeutics(《古德曼&吉尔曼:药理学基础》):在阿司匹林/NSAIDs 与 COX 通路处介绍血栓烷与前列环素的平衡。
Guyton and Hall Textbook of Medical Physiology(《盖顿与霍尔生理学》):在血管张力与止血相关内容中提及血栓烷的作用。
Hamberg M, Svensson J, Samuelsson B. “Thromboxanes: a new group of biologically active compounds derived from prostaglandin endoperoxides.”(1970年代经典论文):早期系统阐述血栓烷的发现与生物学活性。